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Dihexa

N-hexanoic-Tyr-Ile-(6) aminohexanoic amide

Overview

Dihexa is a small peptide-derived compound developed at Washington State University that acts as a potent hepatocyte growth factor (HGF) agonist. It has demonstrated extraordinary potency in cognitive enhancement — reportedly 7 orders of magnitude more potent than BDNF in stimulating new synapse formation making it one of the most potent known cognitive enhancers per molar dose. Dihexa promotes synaptogenesis and has shown remarkable reversal of cognitive deficits in animal models of Alzheimer's disease and age-related cognitive decline. It is one of the most discussed nootropic peptides in the research community despite very limited human data.

Compound Data

Dihexa structure

Molecular Formula

C27H44N4O5

Molecular Weight

504.70 g/mol

IUPAC Name

(2S,3S)-N-(6-amino-6-oxohexyl)-2-[[(2S)-2-(hexanoylamino)-3-(4-hydroxyphenyl)propanoyl]amino]-3-methylpentanamide

PubChem CID

129010512

Where does Dihexa sit?

See how this peptide compares across all 70 peptides in our database.

Mechanism of Action

Dihexa is an agonist of the hepatocyte growth factor (HGF) receptor c-Met. HGF/c-Met signaling in the brain promotes dendritic spine growth axonal outgrowth and synapse formation — the physical substrate of learning and memory. Dihexa promotes the formation of new synaptic connections between neurons in the prefrontal cortex and hippocampus the brain regions most critical for working memory and long-term potentiation. In Alzheimer's disease models Dihexa reversed cognitive deficits associated with amyloid accumulation by restoring synaptic connectivity independent of amyloid clearance. The compound binds to HGF and facilitates its interaction with c-Met with extraordinary efficiency. It is orally bioavailable and crosses the blood-brain barrier making it operationally accessible despite being a peptide-derived compound.

Dosage Information

Typical Dose

10-30 mg

Frequency

Once daily or every other day

Administration

Oral or transdermal

Notes

Very limited human safety data. Extremely potent — start at lowest effective dose. Long duration of action reported anecdotally. Some researchers report effects lasting weeks after discontinuation suggesting durable synaptic changes.

Potential Side Effects

Very limited human safety dataExcessive synaptogenesis theoretical concern with long-term useAnxiety or overstimulation at high doses (reported anecdotally)Headache (uncommon)

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Quick Facts

Administration
Oral or transdermal
Typical Dose
10-30 mg
Frequency
Once daily or every other day
References
0 curated + 4 from PubMed
Evidence Score
13.2 / 100